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Similarly, the TK factor for human variability(HK ) represents the ratio of the doses in average and sensitiveindividuals that would produce the identical internal (targettissue) exposure.

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Thisresidue is within the major site for MDM2 attachment [55] and as a result of itsphosphorylation, p53-MDM2 interaction is disrupted leading to p53 stabilization.This cascade culminates in the G1 phase checkpoint wherein activated p53 inducesthe expression of its direct transcriptional target, p21, causing cell cycle arrest. It is frequently familial but may also be seen in association with otherneurological disorders (282). Lukashchuk N, Vousden KH (2007) Ubiquitination and degradation of mutant p53

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